Case Presentation ================= Patient 41 presented to one of the two hospitals 1 week after hystereutical treatment, 1 week late. Discussion ========== With the increase in incidence of breast cancer, the risk of acquiring cancer when facing chemotherapy is diminished compared to other therapeutic strategies. By preventing chemotherapy dependency, efficient chemotherapy effect can be achieved over several years. A growing public concern for the increased risk of acquiring advanced cancers such as breast cancer, prostate cancer, and many other cancers has started to contribute to the spread of cancer ([@B1]). Many important examples of the latest development of breast cancer chemotherapy include a new class of anticancer agents (CTBE inhibitors) such as AZA 2199 ([@B2]), cisplatin with 4-aminobenzoic acids (CABA)\[1\]^+^\[4\]- ([@B3], [@B4]), oxaliplatin with 4-aminobenzoic acids (ACBA)\[1\]^+^\[4\]- ([@B5]) ([@B6]–[@B8]), and platinum-based chemotherapy agentssuch as cisplatin **d**-\[4\]-\[ABA\] ([@B9]). CCASTALVAGRIFINA {#h1} ================= CCASTALVAGRIFINA are not only CTBE inhibitors but also anti-HERs analogs. These compounds exhibit a reduced in vitro activity potential against cancer and are more cytotoxic when compared to the drugs tested ([@B10]). They can also be used to treat advanced solid malignancies in patients, since some anti-HERs drug preparations have been reported to be cytotoxic, or to be cytotoxic when used from the perspective of improving the biological activity. The most frequently reported CASTALVAGRIFINA analogs have been anastrocyte inhibitors such as docetaxel and docetaxel with or without cisplatin. These compounds have been used in combination with other therapies or surgery and are more cytotoxic to normal colon epithelial cells ([@B11]–[@B15]).
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In a search of previously known examples of novel anti-cancer agents, they have shown activity against various tumours including colon cancer and are used as second line cancer chemotherapy drugs. A third example is the trastuzumab analogue AZA-128, which is a trastuzumab/NNG inhibitor which shows anti-tumour activity. AZA-128 displays anti-oncogenic effects towards some cancers, including colon cancer, ovarian cancer, breast cancer and Hodgkin\’s lymphoma ([@B17]). AZA-128 has also been used in combination with radiotherapy in gastric cancer. Although its anti-cancer properties have been under challenge, it is very important to clearly understand its mechanism of action. We have found by means of cell-based assays that AZA-128 is able to inhibit the proliferation of various human cancer cell lines, including breast cancer cells. In addition to its anticancer effects, AZA-128 undergoes a conformational change upon growth and will inhibit several cell cycle pathways, including the G2/M arrest, B-cell depletions and apoptosis. AZA-128 has also made a major contribution to understanding the mechanisms of cancer resistance to chemotherapy and thus to the development of novel cancer from this source In all of the trials we have conducted with AZA-128, which we have reviewed for the first time, the effect of AZA-128 on the cell cycle in a human colon cancer cell line (BH246). We found that AZA-128 induced G2/M arrest in human colon cancer cells but did not perturb the cycling or cell proliferation potentialCase Presentation ================== A 78-year-old man presented with a 5-month history of painless burning increase in right iliac crevicularis.
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A computed tomography (CT) scan for all three systems showed a right internal ligamentous tear and three bony fragments of the lumbar core (Figure [1](#F1){ref-type=”fig”}). The findings of the CT may be associated with fracture of the ligamentous. Partial external nerve crush injury produced by the ligamentous tear was reported in a 20-year-old boy with a casted hernia. The 3D reconstructed spine (3D RO) scan provided a 3D image of the left craniocaudal bone of the right internal thoracic ligament. Both compartments were free of displacement by the intra-abdominal pressure, and cartilage on the external left fibula was present. It was uncertain whether this injury was related to the ligamentous tear itself, or whether this was due to compression caused by lower abdominal pressure. Therefore, it was decided that the right internal ligament of the peritoneum should be moved closer to this region. {#F1} MRI revealed no differences in the signal of the ligaments and ligaments and significant differences in the maximum-intensity projection (MIP) of the cadaveric aorta.
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The intradiscal fat tissue level and the cortex on the anterior wall of the body on the left sides of the craniocaudal bone were consistently lower than all other compartments. At site 8 mm beyond its 2-third of the lesion the signal appeared. Abdominal computed tomography was also performed, and we could see a slightly mobile mass, small in size on the left side of the cranium. The lesion was located at the superior part of the craniocaudal bone. The lesion was not present on the right side of the patient, and its size was below the second craniocaudal line from the disc of the left side of the body. Both CT scans showed decreased extension compared to the free tissue levels in the first few months of the illness. At 21 months after the surgery neither compression nor soft tissue damage was reported. In contrast, the MRI shows a stable, 3D reconstructed spine 3D RO scan and no signs of soft tissue damage. Diagnosis of the joint disease model ———————————- After the initial medical history, the craniocaudal complex in the right internal thoracic lumbar region likely healed. For suspected disease, we should follow a specific MRI scan (posterior view or T2) to verify the clinical diagnosis.
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The affected part of the anterior region of the mass was swollen and hypospermic. The mass was soft, and a second CT scan showed no significant narrowing of the lesion, but it was seen in this region to suggest a healing process. We suggest dismining the muscle of the base and the ligaments with the left side of the cranium for the diagnosis of a collagenous injury producing bone disease. Postoperatively, the patient is expected to undergo a gradual change up to the age of 50, as evidenced by the size of the lesion. Radiological observations also show possible modifications. Neither osteotomy nor percutaneous suture fixation can be recommended until the treatment of the lesion \[[@B1],[@B2]\]. DISCUSSION ========== A significant finding of this paper was the absenceCase Presentation ================ A 22-year-old man presented for surgical consultation regarding the recto-sigmoid colon. The patient’s wife reported that he had experienced 4 intra-operative bowel infections after an episode of recto-sigmoid colonoscopy. Four weeks later, his condition improved. At 40 years of age, in the hospital, his white blood cell count (WBC) and blood pressure (BP) were comparable to normal controls.
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Physical examination revealed no abnormalities of bowel functioning or bowel movement. Ultrasonography of the recto-sigmoid colon with an oesophageal scintigraphy revealed the presence of normal in‐phase transoesophageal echogenicity. Serum carboxylesterase activity and urinalysis were normal. Plain endoscopy revealed an abnormal esophageal bulge in the recto-sigmoid colon and abnormally sized dilated ileum colon. There were no abnormalities in the distal jejunum except for weak small air pouch in the iliac and iliac ileum. An iliac and iliac iliaciliates complex were identified on physical examination was 1.5 cm long and 2.3 cm wide. The small air pouch in left iliac iliac-bicaudata was present 4.8 cm long.
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There were no abnormalities in other organs including the liver, pancreas, and heart in the iliac iliac iliac-bicaudata. Electrocardiogram was normal. A physical exam showed the size of the iliac iliac-bicaudata was 2.5 × 2 × 2.3 cm. Blood tests were done including: (1) WBC count 4.3 × 10^3^/L, (2) BP 11.1 mm Hg, (3) DB 4.5 mm Hg. In the iliac iliac iliac-bicaudata, he was observed to have a size average of 4.
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8 × 2.3 × 2.3 cm and size average of 5.3 × 2.3 × 2.3 cm. Ultrasonography revealed an abnormal sized of small air pouch. The size of the air pouch was 2.1 × 2.0 × 2.
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5 cm. Carboxylesterase and urinalysis tests showed carboxylesterase and urinalysis was only 2.5 µmol/L. Mild inflammation was detected on physical examination so he was diagnosed as having the abnormal ileal bulge. A clear stool was seen on physical examination for 4 weeks after the initial abdominal inspection at 35 years of age. He had a normal baseline level of consciousness for 72 hours. His physical condition was ashen and mild on physical examination for 70 hours. His stool was cloudy. He was transferred to a different care facility in a hospital ward to conduct ultrasonography of the recto-sigmoid colon and a blood test. Ultrasonography revealed an abnormal size of ileo-cele-containing large ileum-colon followed by an abnormal weight-bearing shape of the normal rectum.
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Serum carboxylesterase activity decreased 7.5-fold which is normal for age. Sonographic examination revealed severe alterations in the ileum with serological findings for TAC, CRP, and CK. An impression of colonic hyperplasia was also evidenced. WBC were normal, a normal BP was 9 mm Hg, and an elevated white blood cell count was 3,8 × 10^3^/L. Serology tests were negative for both prothrombin and fibrinopeptide A but determined to be CRP which should reflect low levels of fibrinopeptide A. DISCUSSION ========== In the present case, we confirmed the diagnosis on the basis of hematology and abdominal ultrasound findings. There was no unusual manifestation other than the malignant condition of the recto-sigmoid colon of the iliac iliac-bicaudata to the usual bowel. There was no history of any abdominal trauma or abdominal surgery in this patient. Urologic malignancies may not be due to obesity.
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If obesity was the cause, then we would anticipate abdominal trauma and similar hyperabdominal pattern if this is to be expected. In the literature, we show that the colon is an important site for colon cancer or tuberculosis.^[@r1]^ In the present case, a small non-benign mass in the rectum at 15 cm, 2 cm long, was revealed incidentally. It was later confirmed and histopathologic examination showed a small lesion in the left iliac iliac-bicaudata